Make sure to cite evidenced based practice guidelines/research in all of your answers as well as on your reference list
1. Jonathan is a 62-year-old who is on angiotensin-converting enzyme (ACE) inhibitor for his newly diagnosed hypertension. He returns to the clinic in a month and his blood pressure has improved from 180/92 on his last visit to 132/82 mm Hg. He denies chest pain, orthostasis, or peripheral edema. Jonathan states he thinks he may be catching a cold because he has a persistent, nonproductive cough and he feels he may be developing a cold sore because his upper lip feels thick, full, and a bit tingly as if he is getting a cold sore. On exam you noted his lungs are CTA and his upper lip appears swollen. 12.5 points
1. Based on the information provided above, discuss your 3 most likely differential diagnoses and their corresponding ICD10 codes & rule each one in and out based on evidence base practices
2. Which of the 3 differential diagnoses you included in Question # 1 is your final or working diagnosis and
Discuss your evidenced-based practice rationale.
3. Discuss and provide your pharmacological treatment plan including any required changes.
4. Complete the following table regarding categories of antihypertensive agents
Classification Name a Specific Drug Usual Dosage Possible side effect
Diuretics
Ca++ Chanel Blockers
ACEs
Peripheral Vasodilators
ARBs
2. From the following categories of medications used to treat hyperlipidemia:
Select 2 and (a) give an example of the medication, (b) a brief statement regarding its mechanism of action (c) usual maintenance dosage, and (d) what specific condition it treats:
HMG-CO-A reductase inhibitors
Fibrates,
Bile acid sequestrants,
Omega 3 fatty acids,
Nicotinic acid,
Red yeast rice
as well as whether there are any gender, race, or age considerations you as an FNP provider would need to consider when prescribing the classification of drug.
Example: HMG-CO-A reductase inhibitors (statin) Simvastatin- 20 -40 mg with evening meal – increases the number of DL receptors in the liver, thus lowering LDL levels